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Endocrine Abstracts (2012) 29 P771

Hospital Philanthropy, Craiova, Romania.


In the thyroid gland pathology, thyroadenitis prominently in their scope, whose etiologic diagnosis is often difficult to objectified and therapeutic attitudes are not yet codified today. mechanisms production in the thyroid thyroid lesions are still far from being elucidated, and concepts are still ongoing reshuffle. There is another explanation that can withstand a critical analysis of how the thyroid’s self tolerance is repealed and the breaking of tolerance in a row followed by antithyroid antibodies and thyroid lesion.

Personal research were performed on 89 cases admitted to the Endocrinology Clinic in the period 2000–2009, whose age ranged from 15–68 years. The diagnosis was suggested by historical data and clinical and laboratory investigations confirmed the etiology of each case. Therapeutic attitudes were conditioned by the results of laboratory assays. He turned to antibiotics, corticosteroids, NSAIDs, antithyroid synthetic thyroid hormones or thyroidotomy.

The results show that the treatment of choice is thyroadenitis administration of glucocorticoid hormones. In subacute thyroiditis phase required a stunning combination of low doses of antithyroid synthetic beta-blockers. To prevent thyroid nodularisation phenomenon, it is desirable thyroid hormone replacement therapy when natural evolution of the disease remit to hypothyroidism. Surgical indication is considered appropriate only for cases resistant to other modes of therapeutic approach to cases abcedate.

Declaration of interest: The authors declare that there is no conflict of interest that could be perceived as prejudicing the impartiality of the research project.

Funding: This research did not receive any specific grant from any funding agency in the public, commercial or not-for-profit sector.

Volume 29

15th International & 14th European Congress of Endocrinology

European Society of Endocrinology 

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