Searchable abstracts of presentations at key conferences in endocrinology

Endocrine Abstracts

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16th European Congress of Endocrinology

Poster Presentations

Endocrine disruptors

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Diabetes therapy
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Endocrine tumours and neoplasia

ea0035p502 | Endocrine disruptors

Acute exposure of BPA from electronic gadgets does not induce oxidative stress in the rat's brain

ea0035p503 | Endocrine disruptors

The reproductive toxicity of butyl benzyl phthalate in male rabbits: the possible protective role of flaxseed

ea0035p504 | Endocrine disruptors

Treatment with kaempferol resulted in the regulation of cell cycle-related and apoptosis-related genes in cancer cell growth caused by triclosan in MCF-7 breast cancer cells

ea0035p505 | Endocrine disruptors

Treatment of prostate cancer cells with phthalate resulted in the upregulation of cyclin D1 and c-myc and downregulation of p21 via estrogen receptor signaling pathway in the progression of LNCaP prostate cancer cells

ea0035p506 | Endocrine disruptors

Treatment of breast cancer cells with triclosan and octylphenol altered the expressions of cyclin D1 and p21 and induced breast tumor masses via an estrogen receptor-dependent signaling pathway in cellular and mouse xenograft models

ea0035p507 | Endocrine disruptors

Transcriptional levels of aryl-hydrocarbon receptor, aryl-hydrocarbon receptor dependent- and cell cycle-related genes were altered by fenhexamid in human ovarian cancer cells expressing estrogen receptors

ea0035p508 | Endocrine disruptors

Transcriptions of cell cycle-related genes were altered by benzylbutyl phthalate and diisobutyl phthalate via an estrogen receptor α signaling pathway in human ovarian cancer cells

ea0035p509 | Endocrine disruptors

Estrogen and bisphenol A affect enamel formation by different signaling pathways

ea0035p510 | Endocrine disruptors

Organometric parameters of rat’s adrenal glands after 60-day administration of sodium benzoate and application of mexidol

ea0035p511 | Endocrine disruptors

The potential role of bisphenol A in the pathogenesis of polycystic ovary syndrome

ea0035p512 | Endocrine disruptors

Bisphenol A disrupts seminoma cell proliferation following an inverted U-shaped non monotonic dose–response curve, due to its greater affinity for GPR30, the non classical membrane G protein-related estrogen receptor, than for ERβ

ea0035p513 | Endocrine disruptors

Di-ethylhexyl-phthalate is metabolised by human thyroid cells and may influence thyroglobulin secretion

Published by BioScientifica

Endocrine Abstracts
ISSN 1470-3947 (print) | ISSN 1479-6848 (online)
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